Oral scurvy and periodontal disease.
Identifieur interne : 000574 ( Main/Exploration ); précédent : 000573; suivant : 000575Oral scurvy and periodontal disease.
Auteurs : L Z Touyz [Canada]Source :
- Journal (Canadian Dental Association) [ 0709-8936 ] ; 1997.
Descripteurs français
- KwdFr :
- Acide ascorbique (administration et posologie), Acide ascorbique (usage thérapeutique), Carence en acide ascorbique (diagnostic), Carence en acide ascorbique (métabolisme), Collagène (métabolisme), Diagnostic différentiel (MeSH), Humains (MeSH), Hémorragie gingivale (diagnostic), Leucocytes (physiologie), Maladies de la bouche (diagnostic), Maladies de la bouche (métabolisme), Maladies de la bouche (traitement médicamenteux), Maladies parodontales (diagnostic), Maladies parodontales (microbiologie), Maladies parodontales (physiopathologie), Monocytes (physiologie), Parodontite (diagnostic), Plaque dentaire (microbiologie), Scorbut (diagnostic), Scorbut (métabolisme), Scorbut (traitement médicamenteux).
- MESH :
- administration et posologie : Acide ascorbique.
- diagnostic : Carence en acide ascorbique, Hémorragie gingivale, Maladies de la bouche, Maladies parodontales, Parodontite, Scorbut.
- microbiologie : Maladies parodontales, Plaque dentaire.
- métabolisme : Carence en acide ascorbique, Collagène, Maladies de la bouche, Scorbut.
- physiologie : Leucocytes, Monocytes.
- physiopathologie : Maladies parodontales.
- traitement médicamenteux : Maladies de la bouche, Scorbut.
- usage thérapeutique : Acide ascorbique.
- Diagnostic différentiel, Humains.
English descriptors
- KwdEn :
- Ascorbic Acid (administration & dosage), Ascorbic Acid (therapeutic use), Ascorbic Acid Deficiency (diagnosis), Ascorbic Acid Deficiency (metabolism), Collagen (metabolism), Dental Plaque (microbiology), Diagnosis, Differential (MeSH), Gingival Hemorrhage (diagnosis), Humans (MeSH), Leukocytes (physiology), Monocytes (physiology), Mouth Diseases (diagnosis), Mouth Diseases (drug therapy), Mouth Diseases (metabolism), Periodontal Diseases (diagnosis), Periodontal Diseases (microbiology), Periodontal Diseases (physiopathology), Periodontitis (diagnosis), Scurvy (diagnosis), Scurvy (drug therapy), Scurvy (metabolism).
- MESH :
- chemical , administration & dosage : Ascorbic Acid.
- chemical , metabolism : Collagen.
- chemical , therapeutic use : Ascorbic Acid.
- diagnosis : Ascorbic Acid Deficiency, Gingival Hemorrhage, Mouth Diseases, Periodontal Diseases, Periodontitis, Scurvy.
- drug therapy : Mouth Diseases, Scurvy.
- metabolism : Ascorbic Acid Deficiency, Mouth Diseases, Scurvy.
- microbiology : Dental Plaque, Periodontal Diseases.
- physiology : Leukocytes, Monocytes.
- physiopathology : Periodontal Diseases.
- Diagnosis, Differential, Humans.
Abstract
Although gingival bleeding is a manifestation of both scurvy and inflammatory periodontal disease, the two conditions are distinctly separate entities. The defective collagen synthesis associated with scurvy also manifests many of the same symptoms as deficient vitamin C physiology, but neither condition is associated with periodontal disease. Unlike scurvy, the various periodontal diseases are caused by oral plaque microorganisms. The body's reaction to these microorganisms is strongly influenced by the compromised functioning of leucocytes and monocytes. Although certain infections and systemic diseases cause gingival bleeding, avitaminosis-C does not cause commonly encountered periodontitis. Vitamin C should not be used for the prophylaxis or cure of periodontal disease in otherwise healthy, well-nourished individuals. A patient with bleeding gingivae warrants referral to a periodontist, oral medicine specialist, or appropriately qualified dentist for examination and treatment.
PubMed: 9433025
Affiliations:
Links toward previous steps (curation, corpus...)
Le document en format XML
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<term>Ascorbic Acid Deficiency (metabolism)</term>
<term>Collagen (metabolism)</term>
<term>Dental Plaque (microbiology)</term>
<term>Diagnosis, Differential (MeSH)</term>
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<term>Humans (MeSH)</term>
<term>Leukocytes (physiology)</term>
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<term>Mouth Diseases (drug therapy)</term>
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<term>Scurvy (metabolism)</term>
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<term>Carence en acide ascorbique (métabolisme)</term>
<term>Collagène (métabolisme)</term>
<term>Diagnostic différentiel (MeSH)</term>
<term>Humains (MeSH)</term>
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<term>Leucocytes (physiologie)</term>
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<term>Maladies de la bouche (métabolisme)</term>
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<term>Maladies parodontales (physiopathologie)</term>
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<term>Scorbut (traitement médicamenteux)</term>
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<term>Gingival Hemorrhage</term>
<term>Mouth Diseases</term>
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<term>Maladies de la bouche</term>
<term>Maladies parodontales</term>
<term>Parodontite</term>
<term>Scorbut</term>
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<term>Scurvy</term>
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<term>Mouth Diseases</term>
<term>Scurvy</term>
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<term>Maladies de la bouche</term>
<term>Scorbut</term>
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<term>Monocytes</term>
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<front><div type="abstract" xml:lang="en">Although gingival bleeding is a manifestation of both scurvy and inflammatory periodontal disease, the two conditions are distinctly separate entities. The defective collagen synthesis associated with scurvy also manifests many of the same symptoms as deficient vitamin C physiology, but neither condition is associated with periodontal disease. Unlike scurvy, the various periodontal diseases are caused by oral plaque microorganisms. The body's reaction to these microorganisms is strongly influenced by the compromised functioning of leucocytes and monocytes. Although certain infections and systemic diseases cause gingival bleeding, avitaminosis-C does not cause commonly encountered periodontitis. Vitamin C should not be used for the prophylaxis or cure of periodontal disease in otherwise healthy, well-nourished individuals. A patient with bleeding gingivae warrants referral to a periodontist, oral medicine specialist, or appropriately qualified dentist for examination and treatment.</div>
</front>
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<Abstract><AbstractText>Although gingival bleeding is a manifestation of both scurvy and inflammatory periodontal disease, the two conditions are distinctly separate entities. The defective collagen synthesis associated with scurvy also manifests many of the same symptoms as deficient vitamin C physiology, but neither condition is associated with periodontal disease. Unlike scurvy, the various periodontal diseases are caused by oral plaque microorganisms. The body's reaction to these microorganisms is strongly influenced by the compromised functioning of leucocytes and monocytes. Although certain infections and systemic diseases cause gingival bleeding, avitaminosis-C does not cause commonly encountered periodontitis. Vitamin C should not be used for the prophylaxis or cure of periodontal disease in otherwise healthy, well-nourished individuals. A patient with bleeding gingivae warrants referral to a periodontist, oral medicine specialist, or appropriately qualified dentist for examination and treatment.</AbstractText>
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